- walking function
Lactobacillus curvatus CP2998
Lactobacillus curvatus CP2998
A lactic acid bacterial strain selected from the Asahi Group’s extensive library of proprietary lactic acid bacteria for its anticipated benefits in improving walking motor function. It is the world’s first lactic acid bacterial strain shown to improve walking motor function in healthy seniors.
Research background
About 25% of the adult population has reportedly not been getting enough exercise in recent years. Inadequate exercise has been cited as a direct risk factor rivaling the importance of factors such as high blood pressure and high cholesterol in determining the likelihood that seniors will need long-term care. While getting regular exercise is a key requirement for promoting and maintaining a healthy lifestyle, efficient methods of improving motor functions also need to be developed.
To meet this need, the Asahi Group has been focusing on discovering materials designed to promote motor function improvement since 2014. Lactobacillus curvatus CP2998 is a lactic acid bacterial strain that inhibits the expression of genes tied to muscle atrophy. It was discovered by drawing on the expertise acquired by the Group from years of studying the functionality of lactic acid bacteria.
Major research findings
- Better walking motor function
- Better vitality and mental health
Creating a society of health and abundance
The problem of Japan’s declining birthrates and aging population continues to gain momentum, calling for efforts to promote healthy life extension that will keep the society healthy and active for as long as possible. While getting exercise is crucial for maintaining health, getting regular exercise can be difficult for seniors or people of any age. Lactobacillus curvatus CP2998 improves walking motor function and can be added to the regular diet to help promote healthy life extension by inhibiting declining motor function.
Research information on Lactobacillus curvatus CP2998
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Paper
Lactobacillus curvatus CP2998 Prevents Dexamethasone-Induced Muscle Atrophy in C2C12 Myotubes